Files

Download

Download Full Text (167 KB)

Download Abstract (202 KB)

Division

East Florida

Hospital

HCA Florida Westside Hospital

Specialty

Internal Medicine

Document Type

Poster

Publication Date

2023

Keywords

hypertensive emergency, multiorgan failure

Disciplines

Cardiovascular Diseases | Internal Medicine | Medicine and Health Sciences

Abstract

Introduction: Hypertensive emergency is defined as a systolic blood pressure greater than 180 mmHg or diastolic pressure over 120 mmHg with signs of end organ damage. The pathophysiology of a hypertensive emergency leads to an inability to maintain tissue perfusion resulting in organ hypoxia that often leads to temporary, and in more severe cases, permanent damage.

Case Description: A 27-year-old gentleman with no known past medical history presented with acute bilateral blurry vision, headache and bilateral lower extremity weakness for the past two days. His blood pressure was 266/169 and heart rate 99 beats per minute on presentation. Bloodwork demonstrated a creatinine level of 6.3 mg/dL, blood urea nitrogen 50 mg/dL, troponin I 0.466 ng/mL, hemoglobin 9.3 g/dL, platelet count 92,000/uL, low haptoglobin and elevated LDH levels. His platelet count was previously within normal limits. Schistocytes were noted on blood smear. His magnetic resonance imaging of the brain showed a 3 mm punctate acute ischemic infarct in the right parietal lobe. He was started on a nicardipine drip and transitioned to oral antihypertensives the following day. Renal function stabilized and he did not require renal replacement therapy. He was discharged on aspirin and a statin, as well as a blood pressure regimen of metoprolol tartrate, hydralazine, and nifedipine.

Discussion: This was a rare presentation of a young male with no past medical history who presented with microangiopathic hemolytic anemia and acute renal failure secondary to a hypertensive emergency. It stood out as the laboratory abnormalities were indicative of microthrombi formation in the systemic vasculature as a result of endothelial damage, similar to more commonly described pathologies such as TTP and HUS.

Acute Multiorgan Failure in a 27-Year-Old Non-Drug User

Share

COinS
 
 

To view the content in your browser, please download Adobe Reader or, alternately,
you may Download the file to your hard drive.

NOTE: The latest versions of Adobe Reader do not support viewing PDF files within Firefox on Mac OS and if you are using a modern (Intel) Mac, there is no official plugin for viewing PDF files within the browser window.